Alzheimer's disease and related disorders annual. / 2004 by Philip Scheltens MD PhD, Serge Gauthier, Philip Scheltens,

By Philip Scheltens MD PhD, Serge Gauthier, Philip Scheltens, Jeffrey L. Cummings

content material: Innervation of Senile Plaques: A hyperlink among Amyloid and Neurofibrillary Pathology? B. Delatour, V. Blanchard, L. Pradier, and C. Duyckaerts Vascular elements and Alzheimer's affliction, I. Skoog, D. Gustafson, and M. Sjogren The function of White topic Lesions in Dementia, L. Pantoni Subclassification of gentle Cognitive Impairment in learn and medical perform, S. Gauthier and J. Touchon Neuropsychiatric features of light Cognitive Impairment, T.J. Hung and J.L. Cummings Cognitive Rehabilitation in gentle Cognitive Impairment and Prodromal Alzheimer's illness, M. van der Linden, A.-C. Juillerat, and X. Delbeuck scientific Trial layout within the Age of Dementia remedies: demanding situations and possibilities, E. Mohr, C.L. Barclay, R. Anderson, and J. consistent Ethics of analysis in Dementia, J. Karlawish Memantine: A Glutamate Antagonist for remedy of Alzheimer's illness, R. Smith Doody, B. Winbald, and V. Jelic administration of Comorbidity in Alzheimer's disorder, R. Jones dietary Prevention in Alzheimer's illness, O. Guerin and B. Vellas Index

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21,29–33 A possiblemechanism for the reported association between hypertension and AD may be that ischemic WMLs increase the possibility that individuals with Alzheimer lesions in their brains express a dementia syndrome. 35 Clinical diagnosis The typical clinical picture of AD is a dementia with insidious onset and a slowly progressive loss of memory and other cognitive functions, affecting language, visuospatial abilities, executive function, orientation, praxis, and behavior. NINCDS-ADRDA criteria36 state that the diagnosis of probable AD requires the absence of systemic disorders or other brain diseases that, alone, could account for the dementia.

Fassbender K, Simons M, Bergmann C et al. Simvastatin strongly reduces levels of Alzheimer’s disease beta-amyloid peptides Abeta 42 and Abeta 40 in vitro and in vivo. Proc Natl Acad Sci USA 2001; 10:10. 121. Refolo LM, Malester B, LaFrancois J et al. Hypercholesterolemia accelerates the Alzheimer’s amyloid pathology in a transgenic mouse model. Neurobiol Dis 2000; 7:321–331. 122. Refolo LM, Pappolla MA, LaFrancois J et al. A cholesterollowering drug reduces beta-amyloid pathology in a transgenic mouse model of Alzheimer’s disease.

In: Swales J, ed. Textbook of Hypertension. Oxford: Blackwell Scientific, 1994; 409–426. Rels D. The brain and hypertension. Arch Neurol 1988; 45:180–183. 40 VASCULAR FACTORS AND ALZHEIMER’S DISEASE 89. Lantos P, Cairns N. The neuropathology of Alzheimer’s disease. In: Levy R, ed. Dementia. London: Chapman and Hall, 1994:185–207. 90. Adams J, Brierley J, Connor R, Treip C. The effects of systemic hypotension upon the human brain. Clinical and neuropathologic observations in 11 cases. Brain 1966; 89:235–267.

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